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Case study global aphasia

Global aphasia is a severe form of nonfluent aphasia, caused by damage to the left side of the brain, that affects [1] receptive and expressive language skills needed for both written and oral language as well as auditory and visual comprehension. The lesion is caused by an occlusion of the left middle cerebral artery [4] [7] and is associated with damage to Broca's area , Wernicke's area , and insular regions which are associated with aspects of language. It is most common for the onset of global aphasia to occur after a thrombotic stroke at the trunk of the middle cerebral artery , with varying severity. Persons with global aphasia are socially appropriate, usually attentive, and task-oriented. If a suspected brain injury has occurred, the patient undergoes a series of medical imaging, which could include MRI magnetic resonance imaging or CT computed tomography scan. The CoBaGa assesses cognitive functions such as attention, executive functions, logical reasoning, memory, visual-auditory recognition, and visual-spatial ability.
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Case Study - Global Aphasia

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Case Study - Global Aphasia Apraxia | The Aphasia Center

Metrics details. For the first time, we estimate the frequency of aphasia following the first-ever ischemic stroke and its relationship to the site of lesion in Arabic-speaking countries. The study was conducted on acute or subacute ischemic stroke patients. Global aphasia was mainly associated with complete middle cerebral artery MCA territories infarction. Motor aphasia was associated mainly with frontal or frontoparietal operculum lesions as well as basal ganglionic lesions. Sensory aphasia was associated with temporal lobe lesions, inferior parietal lobe, and basal ganglia. Conductive aphasia showed lesions in the deep white matter of the frontal and parietal lobes.
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Case Studies

This case study concerns an adult with Wernicke's aphasia characterized by neologistic jargon and a severe auditory comprehension deficit. No communicative or linguistic improvements had been observed after eight months poststroke. Up until this time, therapeutic intervention was aimed at managing the patient's rambling communicative style and improving auditory comprehension skills.
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